Nonalcoholic fatty liver disease: molecular mechanisms for the hepatic steatosis
DC Field | Value | Language |
---|---|---|
dc.contributor.author | Koo, S.-H. | - |
dc.date.accessioned | 2021-09-06T09:41:10Z | - |
dc.date.available | 2021-09-06T09:41:10Z | - |
dc.date.created | 2021-06-17 | - |
dc.date.issued | 2013 | - |
dc.identifier.issn | 2287-2728 | - |
dc.identifier.uri | https://scholar.korea.ac.kr/handle/2021.sw.korea/105862 | - |
dc.description.abstract | Liver plays a central role in the biogenesis of major metabolites including glucose, fatty acids, and cholesterol. Increased incidence of obesity in the modern society promotes insulin resistance in the peripheral tissues in humans, and could cause severe metabolic disorders by inducing accumulation of lipid in the liver, resulting in the progression of non-alcoholic fatty liver disease (NAFLD). NAFLD, which is characterized by increased fat depots in the liver, could precede more severe diseases such as non-alcoholic steatohepatitis (NASH), cirrhosis, and in some cases hepatocellular carcinoma. Accumulation of lipid in the liver can be traced by increased uptake of free fatty acids into the liver, impaired fatty acid beta oxidation, or the increased incidence of de novo lipogenesis. In this review, I would like to focus on the roles of individual pathways that contribute to the hepatic steatosis as a precursor for the NAFLD. | - |
dc.language | English | - |
dc.language.iso | en | - |
dc.subject | acetyl coenzyme A | - |
dc.subject | fatty acid | - |
dc.subject | triacylglycerol | - |
dc.subject | fatty liver | - |
dc.subject | human | - |
dc.subject | lipogenesis | - |
dc.subject | metabolism | - |
dc.subject | mitochondrion | - |
dc.subject | nonalcoholic fatty liver | - |
dc.subject | pathology | - |
dc.subject | Acetyl Coenzyme A | - |
dc.subject | Fatty Acids | - |
dc.subject | Fatty Liver | - |
dc.subject | Humans | - |
dc.subject | Lipogenesis | - |
dc.subject | Mitochondria | - |
dc.subject | Non-alcoholic Fatty Liver Disease | - |
dc.subject | Triglycerides | - |
dc.title | Nonalcoholic fatty liver disease: molecular mechanisms for the hepatic steatosis | - |
dc.type | Article | - |
dc.contributor.affiliatedAuthor | Koo, S.-H. | - |
dc.identifier.doi | 10.3350/cmh.2013.19.3.210 | - |
dc.identifier.scopusid | 2-s2.0-84898439031 | - |
dc.identifier.bibliographicCitation | Clinical and molecular hepatology, v.19, no.3, pp.210 - 215 | - |
dc.relation.isPartOf | Clinical and molecular hepatology | - |
dc.citation.title | Clinical and molecular hepatology | - |
dc.citation.volume | 19 | - |
dc.citation.number | 3 | - |
dc.citation.startPage | 210 | - |
dc.citation.endPage | 215 | - |
dc.type.rims | ART | - |
dc.type.docType | Review | - |
dc.description.journalClass | 1 | - |
dc.description.journalRegisteredClass | scopus | - |
dc.description.journalRegisteredClass | kci | - |
dc.subject.keywordPlus | acetyl coenzyme A | - |
dc.subject.keywordPlus | fatty acid | - |
dc.subject.keywordPlus | triacylglycerol | - |
dc.subject.keywordPlus | fatty liver | - |
dc.subject.keywordPlus | human | - |
dc.subject.keywordPlus | lipogenesis | - |
dc.subject.keywordPlus | metabolism | - |
dc.subject.keywordPlus | mitochondrion | - |
dc.subject.keywordPlus | nonalcoholic fatty liver | - |
dc.subject.keywordPlus | pathology | - |
dc.subject.keywordPlus | Acetyl Coenzyme A | - |
dc.subject.keywordPlus | Fatty Acids | - |
dc.subject.keywordPlus | Fatty Liver | - |
dc.subject.keywordPlus | Humans | - |
dc.subject.keywordPlus | Lipogenesis | - |
dc.subject.keywordPlus | Mitochondria | - |
dc.subject.keywordPlus | Non-alcoholic Fatty Liver Disease | - |
dc.subject.keywordPlus | Triglycerides | - |
dc.subject.keywordAuthor | De novo lipogenesis | - |
dc.subject.keywordAuthor | Fatty acid beta oxidation | - |
dc.subject.keywordAuthor | Free fatty acids | - |
dc.subject.keywordAuthor | TG secretion | - |
Items in ScholarWorks are protected by copyright, with all rights reserved, unless otherwise indicated.
(02841) 서울특별시 성북구 안암로 14502-3290-1114
COPYRIGHT © 2021 Korea University. All Rights Reserved.
Certain data included herein are derived from the © Web of Science of Clarivate Analytics. All rights reserved.
You may not copy or re-distribute this material in whole or in part without the prior written consent of Clarivate Analytics.