GDA-lipase1 (CaGL1) contributes to wound stress resistance by modulation of CaPR-4 expression in hot pepper
DC Field | Value | Language |
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dc.contributor.author | Kim, Ki-Jeong | - |
dc.contributor.author | Lim, Jee Hyuck | - |
dc.contributor.author | Kim, Min Jung | - |
dc.contributor.author | Kim, Taesung | - |
dc.contributor.author | Chung, Hyen Mi | - |
dc.contributor.author | Paek, Kyung-Hee | - |
dc.date.accessioned | 2021-09-09T03:40:28Z | - |
dc.date.available | 2021-09-09T03:40:28Z | - |
dc.date.created | 2021-06-10 | - |
dc.date.issued | 2008-10-03 | - |
dc.identifier.issn | 0006-291X | - |
dc.identifier.uri | https://scholar.korea.ac.kr/handle/2021.sw.korea/122572 | - |
dc.description.abstract | A full length cDNA clone encoding Capsicum annuum GDSL-lipase 1 (CaGL1) was isolated by microarray analysis. The expression of CaGL1 was triggered by methyl jasmonic acid (MeJA), an important signal in abiotic/biotic stress response. However, the expression of this gene was not increased by the application of salicylic acid (SA) or ethylene treatment. And, local/systemic Wounding stimuli resulted in rapid accumulation of CaGL1 mRNA. However, CaGL1 was not specifically induced during the hypersensitive response upon Tobacco mosaic virus (TMV) inoculation. By using a virus-induced gene silencing (VIGS)-based reverse genetic approach, it was observed that the suppression of CaGL1 attenuates the expression of Capsicum annuum pathogenesis-related protein 4 (CaPR-4) during wound stress. However, the CaPR-4 transcript level induced by TMV was not regulated by CaGL1 expression. These results indicate that CaGL1 may be involved in signaling pathway of MeJA and/or the wound responses through CaPR-4 expression modulation. (c) 2008 Elsevier Inc. All rights reserved. | - |
dc.language | English | - |
dc.language.iso | en | - |
dc.publisher | ACADEMIC PRESS INC ELSEVIER SCIENCE | - |
dc.subject | TOBACCO-MOSAIC-VIRUS | - |
dc.subject | GENE | - |
dc.subject | LIPASE | - |
dc.subject | ARABIDOPSIS | - |
dc.subject | PROTEIN | - |
dc.subject | DEFENSE | - |
dc.subject | ENZYMES | - |
dc.subject | TOMATO | - |
dc.subject | RICE | - |
dc.title | GDA-lipase1 (CaGL1) contributes to wound stress resistance by modulation of CaPR-4 expression in hot pepper | - |
dc.type | Article | - |
dc.contributor.affiliatedAuthor | Paek, Kyung-Hee | - |
dc.identifier.doi | 10.1016/j.bbrc.2008.07.120 | - |
dc.identifier.scopusid | 2-s2.0-49649089052 | - |
dc.identifier.wosid | 000258866900019 | - |
dc.identifier.bibliographicCitation | BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, v.374, no.4, pp.693 - 698 | - |
dc.relation.isPartOf | BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS | - |
dc.citation.title | BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS | - |
dc.citation.volume | 374 | - |
dc.citation.number | 4 | - |
dc.citation.startPage | 693 | - |
dc.citation.endPage | 698 | - |
dc.type.rims | ART | - |
dc.type.docType | Article | - |
dc.description.journalClass | 1 | - |
dc.description.journalRegisteredClass | scie | - |
dc.description.journalRegisteredClass | scopus | - |
dc.relation.journalResearchArea | Biochemistry & Molecular Biology | - |
dc.relation.journalResearchArea | Biophysics | - |
dc.relation.journalWebOfScienceCategory | Biochemistry & Molecular Biology | - |
dc.relation.journalWebOfScienceCategory | Biophysics | - |
dc.subject.keywordPlus | TOBACCO-MOSAIC-VIRUS | - |
dc.subject.keywordPlus | GENE | - |
dc.subject.keywordPlus | LIPASE | - |
dc.subject.keywordPlus | ARABIDOPSIS | - |
dc.subject.keywordPlus | PROTEIN | - |
dc.subject.keywordPlus | DEFENSE | - |
dc.subject.keywordPlus | ENZYMES | - |
dc.subject.keywordPlus | TOMATO | - |
dc.subject.keywordPlus | RICE | - |
dc.subject.keywordAuthor | CaGL1 | - |
dc.subject.keywordAuthor | CaPR-4 | - |
dc.subject.keywordAuthor | hot pepper (Cupsicum annuum L.) | - |
dc.subject.keywordAuthor | MeJA | - |
dc.subject.keywordAuthor | TMV | - |
dc.subject.keywordAuthor | virus-incluced gene silencing | - |
dc.subject.keywordAuthor | wound | - |
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