Improved Endothelial Function in Patients with Atrial Fibrillation through Maintenance of Sinus Rhythm by Successful Catheter Ablation
DC Field | Value | Language |
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dc.contributor.author | Shin, Seung Yong | - |
dc.contributor.author | Na, Jin Oh | - |
dc.contributor.author | Lim, Hong Euy | - |
dc.contributor.author | Choi, Cheol Ung | - |
dc.contributor.author | Choi, Jong Il | - |
dc.contributor.author | Kim, Seong Hwan | - |
dc.contributor.author | Kim, Eung Ju | - |
dc.contributor.author | Park, Sang Weon | - |
dc.contributor.author | Rha, Seung-Woon | - |
dc.contributor.author | Park, Chang Gyu | - |
dc.contributor.author | Seo, Hong Seog | - |
dc.contributor.author | Oh, Dong Joo | - |
dc.contributor.author | Kim, Young-Hoon | - |
dc.date.accessioned | 2021-09-07T13:32:42Z | - |
dc.date.available | 2021-09-07T13:32:42Z | - |
dc.date.created | 2021-06-14 | - |
dc.date.issued | 2011-04 | - |
dc.identifier.issn | 1045-3873 | - |
dc.identifier.uri | https://scholar.korea.ac.kr/handle/2021.sw.korea/112710 | - |
dc.description.abstract | Methods: This study prospectively enrolled 80 subjects who underwent CA for AF (paroxysmal AF = 61, persistent AF = 19). Eighty subjects with no history of AF were enrolled as controls, all of whom were matched by age, gender, body mass index, and atherosclerotic risk factor distribution. Brachial artery flow-mediated dilatation (FMD) was measured at baseline, and at 1 month and 6 months post CA in AF subjects who remained in SR. Among controls, FMD was measured at baseline and at 6 months. We used high sensitivity C-reactive protein (hs-CRP), interleukin-6, soluble E- or P-selectin, and endothelin-1 as biomarker indices for inflammation and/or ED. Results: Compared with controls, AF subjects had lower FMD at baseline (FMDbaseline, P < 0.001). After successful CA, FMD was significantly improved at 1 month and 6 months, nearly approaching control levels. A multivariate analysis revealed that FMDbaseline, hs-CRP, and left atrial volume (LAV) were independent predictors for arrhythmia recurrence after CA. Other biomarkers were not related to rhythm outcome. Conclusion: AF subjects have significantly impaired FMD, which can be reversed through maintenance of SR by successful CA. FMDbaseline, hs-CRP, and LAV are important predictors for AF recurrence after CA. (J Cardiovasc Electrophysiol, Vol. 22, pp. 376-382). | - |
dc.language | English | - |
dc.language.iso | en | - |
dc.publisher | WILEY-BLACKWELL | - |
dc.subject | C-REACTIVE PROTEIN | - |
dc.subject | VON-WILLEBRAND-FACTOR | - |
dc.subject | FLOW-MEDIATED DILATATION | - |
dc.subject | CORONARY-ARTERY-DISEASE | - |
dc.subject | NITRIC-OXIDE SYNTHASE | - |
dc.subject | SOLUBLE E-SELECTIN | - |
dc.subject | ELECTRICAL CARDIOVERSION | - |
dc.subject | INFLAMMATORY MARKERS | - |
dc.subject | PLATELET ACTIVATION | - |
dc.subject | MECHANISMS | - |
dc.title | Improved Endothelial Function in Patients with Atrial Fibrillation through Maintenance of Sinus Rhythm by Successful Catheter Ablation | - |
dc.type | Article | - |
dc.contributor.affiliatedAuthor | Na, Jin Oh | - |
dc.contributor.affiliatedAuthor | Lim, Hong Euy | - |
dc.contributor.affiliatedAuthor | Choi, Cheol Ung | - |
dc.contributor.affiliatedAuthor | Choi, Jong Il | - |
dc.contributor.affiliatedAuthor | Kim, Seong Hwan | - |
dc.contributor.affiliatedAuthor | Kim, Eung Ju | - |
dc.contributor.affiliatedAuthor | Rha, Seung-Woon | - |
dc.contributor.affiliatedAuthor | Park, Chang Gyu | - |
dc.contributor.affiliatedAuthor | Seo, Hong Seog | - |
dc.contributor.affiliatedAuthor | Oh, Dong Joo | - |
dc.contributor.affiliatedAuthor | Kim, Young-Hoon | - |
dc.identifier.doi | 10.1111/j.1540-8167.2010.01919.x | - |
dc.identifier.scopusid | 2-s2.0-79954569810 | - |
dc.identifier.wosid | 000289470700002 | - |
dc.identifier.bibliographicCitation | JOURNAL OF CARDIOVASCULAR ELECTROPHYSIOLOGY, v.22, no.4, pp.376 - 382 | - |
dc.relation.isPartOf | JOURNAL OF CARDIOVASCULAR ELECTROPHYSIOLOGY | - |
dc.citation.title | JOURNAL OF CARDIOVASCULAR ELECTROPHYSIOLOGY | - |
dc.citation.volume | 22 | - |
dc.citation.number | 4 | - |
dc.citation.startPage | 376 | - |
dc.citation.endPage | 382 | - |
dc.type.rims | ART | - |
dc.type.docType | Article | - |
dc.description.journalClass | 1 | - |
dc.description.journalRegisteredClass | scie | - |
dc.description.journalRegisteredClass | scopus | - |
dc.relation.journalResearchArea | Cardiovascular System & Cardiology | - |
dc.relation.journalWebOfScienceCategory | Cardiac & Cardiovascular Systems | - |
dc.subject.keywordPlus | C-REACTIVE PROTEIN | - |
dc.subject.keywordPlus | VON-WILLEBRAND-FACTOR | - |
dc.subject.keywordPlus | FLOW-MEDIATED DILATATION | - |
dc.subject.keywordPlus | CORONARY-ARTERY-DISEASE | - |
dc.subject.keywordPlus | NITRIC-OXIDE SYNTHASE | - |
dc.subject.keywordPlus | SOLUBLE E-SELECTIN | - |
dc.subject.keywordPlus | ELECTRICAL CARDIOVERSION | - |
dc.subject.keywordPlus | INFLAMMATORY MARKERS | - |
dc.subject.keywordPlus | PLATELET ACTIVATION | - |
dc.subject.keywordPlus | MECHANISMS | - |
dc.subject.keywordAuthor | atrial fibrillation | - |
dc.subject.keywordAuthor | catheter ablation | - |
dc.subject.keywordAuthor | C-reactive protein | - |
dc.subject.keywordAuthor | endothelial function | - |
dc.subject.keywordAuthor | inflammation | - |
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