Emerging roles of 14-3-3 gamma in the brain disorder
DC Field | Value | Language |
---|---|---|
dc.contributor.author | Cho, Eunsil | - |
dc.contributor.author | Park, Jae-Yong | - |
dc.date.accessioned | 2021-08-30T09:53:53Z | - |
dc.date.available | 2021-08-30T09:53:53Z | - |
dc.date.created | 2021-06-19 | - |
dc.date.issued | 2020-10-31 | - |
dc.identifier.issn | 1976-6696 | - |
dc.identifier.uri | https://scholar.korea.ac.kr/handle/2021.sw.korea/52087 | - |
dc.description.abstract | 14-3-3 proteins are mostly expressed in the brain and are closely involved in numerous brain functions and various brain disorders. Among the isotypes of the 14-3-3 proteins, 14-3-3 gamma is mainly expressed in neurons and is highly produced during brain development, which could indicate that it has a significance in neural development. Furthermore, the distinctive levels of temporally and locally regulated 14-3-3 gamma expression in various brain disorders suggest that it could play a substantial role in brain plasticity of the diseased states. In this review, we introduce the various brain disorders reported to be involved with 14-3-3 gamma, and summarize the changes of 14-3-3 gamma expression in each brain disease. We also discuss the potential of 14-3-3 gamma for treatment and the importance of research on specific 14-3-3 isotypes for an effective therapeutic approach. | - |
dc.language | English | - |
dc.language.iso | en | - |
dc.publisher | KOREAN SOCIETY BIOCHEMISTRY & MOLECULAR BIOLOGY | - |
dc.subject | CREUTZFELDT-JAKOB-DISEASE | - |
dc.subject | NEURONAL MIGRATION DELAY | - |
dc.subject | CEREBROSPINAL-FLUID | - |
dc.subject | STRUCTURAL BASIS | - |
dc.subject | CELL-CYCLE | - |
dc.subject | IN-VIVO | - |
dc.subject | ALZHEIMERS-DISEASE | - |
dc.subject | SUBCELLULAR-LOCALIZATION | - |
dc.subject | 14-3-3GAMMA PROTEIN | - |
dc.subject | MOLECULAR-CLONING | - |
dc.title | Emerging roles of 14-3-3 gamma in the brain disorder | - |
dc.type | Article | - |
dc.contributor.affiliatedAuthor | Park, Jae-Yong | - |
dc.identifier.doi | 10.5483/BMBRep.2020.53.10.158 | - |
dc.identifier.scopusid | 2-s2.0-85094933227 | - |
dc.identifier.wosid | 000587737700002 | - |
dc.identifier.bibliographicCitation | BMB REPORTS, v.53, no.10, pp.500 - 511 | - |
dc.relation.isPartOf | BMB REPORTS | - |
dc.citation.title | BMB REPORTS | - |
dc.citation.volume | 53 | - |
dc.citation.number | 10 | - |
dc.citation.startPage | 500 | - |
dc.citation.endPage | 511 | - |
dc.type.rims | ART | - |
dc.type.docType | Review | - |
dc.identifier.kciid | ART002639220 | - |
dc.description.journalClass | 1 | - |
dc.description.journalRegisteredClass | scie | - |
dc.description.journalRegisteredClass | scopus | - |
dc.description.journalRegisteredClass | kci | - |
dc.relation.journalResearchArea | Biochemistry & Molecular Biology | - |
dc.relation.journalWebOfScienceCategory | Biochemistry & Molecular Biology | - |
dc.subject.keywordPlus | CREUTZFELDT-JAKOB-DISEASE | - |
dc.subject.keywordPlus | NEURONAL MIGRATION DELAY | - |
dc.subject.keywordPlus | CEREBROSPINAL-FLUID | - |
dc.subject.keywordPlus | STRUCTURAL BASIS | - |
dc.subject.keywordPlus | CELL-CYCLE | - |
dc.subject.keywordPlus | IN-VIVO | - |
dc.subject.keywordPlus | ALZHEIMERS-DISEASE | - |
dc.subject.keywordPlus | SUBCELLULAR-LOCALIZATION | - |
dc.subject.keywordPlus | 14-3-3GAMMA PROTEIN | - |
dc.subject.keywordPlus | MOLECULAR-CLONING | - |
dc.subject.keywordAuthor | 14-3-3 gamma | - |
dc.subject.keywordAuthor | 14-3-3 protein | - |
dc.subject.keywordAuthor | 14-3-3 gamma | - |
dc.subject.keywordAuthor | Brain disorders | - |
dc.subject.keywordAuthor | Glioma | - |
dc.subject.keywordAuthor | Inflammatory disease | - |
dc.subject.keywordAuthor | Neurodegenerative disease | - |
dc.subject.keywordAuthor | Neurodevelopmental disease | - |
dc.subject.keywordAuthor | YWHAG | - |
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